Juvenile Huntington's Disease Skin Fibroblasts Respond with Elevated Parkin Level and Increased Proteasome Activity as a Potential Mechanism to Counterbalance the Pathological Consequences of Mutant Huntingtin Protein.

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dc.contributor.authorAladdin, Azzam
dc.contributor.departmentDE--Általános Orvostudományi Karhu_HU
dc.date.accessioned2020-04-27T09:53:35Z
dc.date.available2020-04-27T09:53:35Z
dc.date.issued2019-10-26
dc.description.etrInt. J. Mol. Sci. 2019, 20(21), 5338hu_HU
dc.identifier.jtitleInternational Journal of Molecular Scienceshu_HU
dc.identifier.urihttp://hdl.handle.net/2437/284924
dc.titleJuvenile Huntington's Disease Skin Fibroblasts Respond with Elevated Parkin Level and Increased Proteasome Activity as a Potential Mechanism to Counterbalance the Pathological Consequences of Mutant Huntingtin Protein.hu_HU
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