Juvenile Huntington's Disease Skin Fibroblasts Respond with Elevated Parkin Level and Increased Proteasome Activity as a Potential Mechanism to Counterbalance the Pathological Consequences of Mutant Huntingtin Protein.
dc.contributor.author | Aladdin, Azzam | |
dc.contributor.department | DE--Általános Orvostudományi Kar | hu_HU |
dc.date.accessioned | 2020-04-27T09:53:35Z | |
dc.date.available | 2020-04-27T09:53:35Z | |
dc.date.issued | 2019-10-26 | |
dc.description.etr | Int. J. Mol. Sci. 2019, 20(21), 5338 | hu_HU |
dc.identifier.jtitle | International Journal of Molecular Sciences | hu_HU |
dc.identifier.uri | http://hdl.handle.net/2437/284924 | |
dc.title | Juvenile Huntington's Disease Skin Fibroblasts Respond with Elevated Parkin Level and Increased Proteasome Activity as a Potential Mechanism to Counterbalance the Pathological Consequences of Mutant Huntingtin Protein. | hu_HU |